Turnover of C99 is controlled by a crosstalk between ERAD and ubiquitin-independent lysosomal degradation in human neuroglioma cells.
Hianara A Bustamante, Andrés Rivera-Dictter, Viviana A Cavieres and 5 more
PLoS ONE · 2013
doi:10.1371/journal.pone.0083096 (opens in a new tab)
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