Acute kidney injury in SARS-CoV-2 infected patients

Abstract

Severe acute respiratory syndrome coronavirus-2 (SARSCoV-2) is the pathogen responsible of atypical pneumonia that has affected more than 330,000 people and caused death of more than 14,000 patients (as of March 23, 2020, WHO Report). Coronavirus spike (S) glycoproteins mediate entry into cells end predominantly lung epithelial alveolar cells precipitating an interstitial pneumonia that evolves through acute respiratory distress syndrome (ARDS) [1].

Cited by papers we hold 5

• Renal complications in coronavirus disease 2019: a systematic review · 2020
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• Repurposing existing drugs for COVID-19: an endocrinology perspective · 2020
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• Hypertension, Thrombosis, Kidney Failure, and Diabetes: Is COVID-19 an Endothelial Disease? A Comprehensive Evaluation of Clinical and Basic Evidence · 2020
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• Cathepsin L in COVID-19: From Pharmacological Evidences to Genetics · 2020
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• Body Localization of ACE-2: On the Trail of the Keyhole of SARS-CoV-2 · 2020
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