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Absence of amyloid β oligomers at the postsynapse and regulated synaptic Zn<sup>2+</sup> in cognitively intact aged individuals with Alzheimer’s disease neuropathology
Bjorklund Nicole L, Reese Lindsay C, Sadagoparamanujam V-M and 3 more
Molecular Neurodegeneration · 2012
References 76
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Cited by papers we hold 6
- Cellular zinc metabolism and zinc signaling: from biological functions to diseases and therapeutic targets · 2024No notice
- Increased excitatory to inhibitory synaptic ratio in parietal cortex samples from individuals with Alzheimer’s disease · 2021No notice
- Challenges for Alzheimer's Disease Therapy: Insights from Novel Mechanisms Beyond Memory Defects · 2018No notice
- Aging and Alzheimer’s disease: Comparison and associations from molecular to system level · 2018No notice
- Soluble amyloid-β oligomers as synaptotoxins leading to cognitive impairment in Alzheimer’s disease · 2015No notice
- Tau Oligomers: The Toxic Player at Synapses in Alzheimer’s Disease · 2015No notice
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