Perinatal Hypoxic‐Ischemic Encephalopathy

Abstract

Perinatal hypoxic‐ischemic encephalopathy (HIE) is an important cause of brain injury in the newborn and can result in long‐term devastating consequences. Perinatal hypoxia is a vital cause of long‐term neurologic complications varying from mild behavioural deficits to severe seizure, mental retardation, and/or cerebral palsy in the newborn. In the mammalian developing brain, ongoing research into pathophysiological mechanism of neuronal injury and therapeutic strategy after perinatal hypoxia is still limited. With the advent of promising therapy of hypothermia in HIE, this paper reviews the pathophysiology of HIE and the future potential neuroprotective strategies for clinical potential for hypoxia sufferers.

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Cited by papers we hold 8

• NF-κB as an Inducible Regulator of Inflammation in the Central Nervous System · 2024
  No notice
• Cell-based treatment for perinatal hypoxic-ischemic encephalopathy · 2021
  Not checked
• HBO Promotes the Differentiation of Neural Stem Cells via Interactions Between the Wnt3/β-Catenin and BMP2 Signaling Pathways · 2019
  Not checked
• Neonatal Hypoxia Ischaemia: Mechanisms, Models, and Therapeutic Challenges · 2017
  No notice
• Sexually dimorphic outcomes and inflammatory responses in hypoxic-ischemic encephalopathy · 2015
  No notice
• Neuroprotective Strategies after Neonatal Hypoxic Ischemic Encephalopathy · 2015
  No notice
• Perinatal hypoxic-ischemic encephalopathy: severity determinants and outcomes · 2014
  Not checked
• Early neural disruption and auditory processing outcomes in rodent models: Implications for developmental language disability · 2013
  Not checked

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