BH4 domain of antiapoptotic Bcl-2 family members closes voltage-dependent anion channel and inhibits apoptotic mitochondrial changes and cell death

Cited by papers we hold 8

• VDAC1 at the Intersection of Cell Metabolism, Apoptosis, and Diseases · 2020
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• Emerging molecular mechanisms in chemotherapy: Ca2+ signaling at the mitochondria-associated endoplasmic reticulum membranes · 2018
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• Voltage-Dependent Anion Channel 1 As an Emerging Drug Target for Novel Anti-Cancer Therapeutics · 2017
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• VDAC1 at the crossroads of cell metabolism, apoptosis and cell stress · 2017
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• Mitochondrial VDAC1: A Key Gatekeeper as Potential Therapeutic Target · 2017
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• Mcl-1 promotes lung cancer cell migration by directly interacting with VDAC to increase mitochondrial Ca2+ uptake and reactive oxygen species generation · 2014
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• VDAC1: from structure to cancer therapy · 2012
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• The evolutionary history of mitochondrial porins · 2007
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