β-arrestin-2 regulates NMDA receptor function in spinal lamina II neurons and duration of persistent pain
Gang Chen, Rou-Gang Xie, Yong-Jing Gao and 8 more
Nature Communications · 2016
doi:10.1038/ncomms12531 (opens in a new tab)
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Abstract
The cellular mechanisms underlying acute pain transitions to chronic pain are poorly understood. Here the authors show that the scaffolding protein β-arrestin 2 contributes to these processes via desensitization of NMDA receptors in spinal neurons.
Abstract licence: CC BY-NC-ND
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