miR-93 regulates Msk2-mediated chromatin remodelling in diabetic nephropathy
Shawn S. Badal, Yin Wang, Jianyin Long and 6 more
Nature Communications · 2016
doi:10.1038/ncomms12076 (opens in a new tab)
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Abstract
Podocyte injury is central to kidney dysfunction in diabetic nephropathy. Here the authors show that Msk2 is a target of miR-93 and this interaction mediates pathogenic chromatin remodelling in diabetic nephropathy.
Abstract licence: CC BY-NC-ND
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