Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: the effects of CaMKII and angiotensin AT1 receptor inhibition
Tomas Rajtik, Slavka Carnicka, Adrian Szobi and 7 more
Data in Brief · 2016
doi:10.1016/j.dib.2016.03.017 (opens in a new tab)
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Abstract
Content of particular proteins indicating cellular injury due to apoptosis and necrosis has been investigated in ischemic/reperfused (IR) hearts and ischemic/reperfused hearts treated with CaMKII inhibitor and/or AT1 receptor inhibitor. This data article provides information in support of the original research article “Oxidative activation of CaMKIIδ in acute myocardial ischemia/reperfusion injury: a role of angiotensin AT1 receptor-NOX2 signaling axis” [1]. Keywords: Myocardial ischemia/reperfusion injury, CaMKII inhibition, AT1 receptor blockade, Apoptosis, Necrosis
Abstract licence: CC BY-NC
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