The Neutrophil NLRC4 Inflammasome Selectively Promotes IL-1β Maturation without Pyroptosis during Acute Salmonella Challenge

Cited by papers we hold 16

• Pyroptosis: molecular mechanisms and roles in disease · 2025
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• The role of NOD-like receptors in innate immunity · 2023
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• NLRP3 Inflammasome Assembly in Neutrophils Is Supported by PAD4 and Promotes NETosis Under Sterile Conditions · 2021
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• Neutrophils: Many Ways to Die · 2021
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• Necroptosis, pyroptosis and apoptosis: an intricate game of cell death · 2021
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• N-GSDMD trafficking to neutrophil organelles facilitates IL-1β release independently of plasma membrane pores and pyroptosis · 2020
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• Interactions Between the Gut Microbiota and the Host Innate Immune Response Against Pathogens · 2019
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• Oxidative Stress and NLRP3-Inflammasome Activity as Significant Drivers of Diabetic Cardiovascular Complications: Therapeutic Implications · 2018
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• Pattern Recognition Receptors and the Host Cell Death Molecular Machinery · 2018
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• The NLRP3 and Pyrin Inflammasomes: Implications in the Pathophysiology of Autoinflammatory Diseases · 2017
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• Reactive Oxygen Species Localization Programs Inflammation to Clear Microbes of Different Size · 2017
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• Inflammasome-dependent IL-1β release depends upon membrane permeabilisation · 2016
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• Neutrophil P2X7 receptors mediate NLRP3 inflammasome-dependent IL-1β secretion in response to ATP · 2016
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• Neutrophils mediate Salmonella Typhimurium clearance through the GBP4 inflammasome-dependent production of prostaglandins · 2016
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• Translocation of interleukin-1β into a vesicle intermediate in autophagy-mediated secretion · 2015
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• Human caspase-4 and caspase-5 regulate the one-step non-canonical inflammasome activation in monocytes · 2015
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