Intrinsic Membrane Hyperexcitability of Amyotrophic Lateral Sclerosis Patient-Derived Motor Neurons

Cited by papers we hold 12

• Human neuronal networks on micro-electrode arrays are a highly robust tool to study disease-specific genotype-phenotype correlations in vitro · 2021
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• Human sensorimotor organoids derived from healthy and amyotrophic lateral sclerosis stem cells form neuromuscular junctions · 2021
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• Neurodegenerative Diseases – Is Metabolic Deficiency the Root Cause? · 2020
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• Repeat‐associated non‐AUG translation in C9orf72‐ALS/FTD is driven by neuronal excitation and stress · 2019
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• Motor Neuron Susceptibility in ALS/FTD · 2019
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• C9ORF72 repeat expansion causes vulnerability of motor neurons to Ca2+-permeable AMPA receptor-mediated excitotoxicity · 2018
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• Kir4.1-Dependent Astrocyte-Fast Motor Neuron Interactions Are Required for Peak Strength · 2018
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• Hypoexcitability precedes denervation in the large fast-contracting motor units in two unrelated mouse models of ALS · 2018
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• HDAC6 inhibition reverses axonal transport defects in motor neurons derived from FUS-ALS patients · 2017
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• Motor neuron vulnerability and resistance in amyotrophic lateral sclerosis · 2017
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• The Unfolded Protein Response and the Role of Protein Disulfide Isomerase in Neurodegeneration · 2016
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• Human iPSC-derived motoneurons harbouring TARDBP or C9ORF72 ALS mutations are dysfunctional despite maintaining viability · 2015
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